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尹艳玲,女,博士,首都医科大学神经生物学系教授、博士生导师。于哈尔滨医科大学获得学士学位,于首都医科大学分别获得硕士学位、博士学位,曾在清华大学生物系、北京大学分子医学研究所参与合作研究。近年来主持国家自然科学基金面上项目、北京市自然科学基金等多项省部级课题,致力于急性缺血性脑卒中发病及干预机制研究,相关成果发表在领域内的top期刊,被脑科学日报等多家媒体报道。
[1] 主持北京市自然科学基金面上项目1项,项目名称:急性脑缺血所致SNAP29蛋白减少对小鼠认知功能影响及其分子机制研究(7202006).
[2] 主持国家自然科学基金面上项目1项,项目名称:急性缺血性脑卒中小鼠皮层神经元自噬底物蛋白p62对神经元焦亡的调控机制研究(31771292).
[3] 主持国家自然科学基金面上项目1项,项目名称:PKC亚型特异性蛋白对缺血性脑卒中小鼠FUNDC1介导线粒体自噬的调节机制研究(31571162).
[4] 主持国家自然科学基金青年基金1项,项目名称:cPKCgamma-CRMP2信号通路在脑缺血低氧性损伤和适应中作用机制的研究(31200895).
[5] 主持北京市优秀人才项目1项,项目名称:脑衰反应蛋白2在脑缺血/低氧性损伤中作用机制的研究(2013D005018000010).
[1] 主持北京市自然科学基金面上项目1项,项目名称:急性脑缺血所致SNAP29蛋白减少对小鼠认知功能影响及其分子机制研究(7202006).
[2] 主持国家自然科学基金面上项目1项,项目名称:急性缺血性脑卒中小鼠皮层神经元自噬底物蛋白p62对神经元焦亡的调控机制研究(31771292).
[3] 主持国家自然科学基金面上项目1项,项目名称:PKC亚型特异性蛋白对缺血性脑卒中小鼠FUNDC1介导线粒体自噬的调节机制研究(31571162).
[4] 主持国家自然科学基金青年基金1项,项目名称:cPKCgamma-CRMP2信号通路在脑缺血低氧性损伤和适应中作用机制的研究(31200895).
[5] 主持北京市优秀人才项目1项,项目名称:脑衰反应蛋白2在脑缺血/低氧性损伤中作用机制的研究(2013D005018000010).
[1] Rongqi Wan, Jiahui Fan, Huimeng Song, WeiSun*, Yanling Yin*. Oxygen-Glucose Deprivation/Reperfusion-InducedSirt3 Reduction Facilitated Neuronal Injuries in an Apoptosis-Dependent MannerDuring Prolonged Reperfusion. NeurochemicalResearch. 2022;47(4):1012-1024. doi: 10.1007/s11064-021-03502-y.
[2] WeijieYan, Jiahui Fan, Xia Zhang, Huimeng Song, Rongqi Wan, Wei Wang*, YanlingYin*. Decreased neuronal synaptosome associated protein 29 contributesto poststroke cognitive impairment by disrupting presynaptic maintenance. Theranostics. 2021,11(10):4616-4636. doi: 10.7150/thno.54210. eCollection 2021
[3] Xia Zhang#, MengpingWei#, Jiahui Fan, Weijie Yan, Xu Zha, Huimeng Song, Rongqi Wan, YanlingYin*, Wei Wang*. Ischemia-induced Upregulation of Autophagy PreludesDysfunctional Lysosomal Storage and Associated Synaptic Impairments in Neurons.Autophagy, 2021Jun;17(6):1519-1542. doi: 10.1080/15548627.2020.1840796. 封面论文
[4] WeijieYan#, Wei Sun#, Jiahui Fan, Haiqing Wang, Song Han, Junfa Li, Yanling Yin*. Sirt1-ROS-TRAF6Signaling-Induced Pyroptosis Contributes to Early Injury in Ischemic Mice. Neuroscience Bulletin. 2020 Aug;36(8):845-859. doi: 10.1007/s12264-020-00489-4.
[5] Yu Diao#, Weijie Yan#, Wei Sun, Yanlin Luo, Junfa Li, Yanling Yin*. The dual role of KCNQ/M channels upon OGD or OGD/R insults in culturedcortical neurons of mice: Timing is crucial in targeting M-channels againstischemic injuries. Journal of Cellular Physiology. 2019, 234(10):12714-12726. doi: 10.1002/jcp.27889. Epub2018 Dec 6. 封面论文
[6] Yun Li, Wei Sun, Song Han, Jianing Li, ShuDing, Wei Wang, Yanling Yin*. IGF-1-involved negativefeedback of NR2B NMDA subunits protects cultured hippocampal neurons againstNMDA-induced excitotoxicity. MolecularNeurobiology. 2017, 54(1),684–696. doi:10.1007/s12035-015-9647-7. Epub 2016 Jan 13.
[7] Yanling Yin*,Yuanyuan Zhao, Song Han, Nan Zhang, Hanyu Chen, Xiaomin Wang*. Autophagy-ERK1/2-involvedDisinhibition of Hippocampal Neurons Contributes to the Pre-synaptic ToxicityInduced by Aß42 Exposure. Journal ofAlzheimers Disease. 2017,59(3): 851–869. doi: 10.3233/JAD-170246.
[8] Yanling Yin,Peng Huang, Zhu Han, Guojun Wei, Changwei Zhou, Jian Wen, Bo Su, Xiaoqin Wang,Yansong Wang. Collagen nanofibers facilitated pre-synaptic maturation in differentiatedneurons from spinal cord-derived neural stem cells through MAPK/ERK1/2-Synapsin I signaling pathway. Biomacromolecules.15(2014):2449-60. (5-yearIF: 6.95) doi: 10.1021/bm500321h. Epub 2014 Jul 1.
[9] YansongWang#, Wei Wang#, Dongguo Li#, Mi Li, Peipei Wang, Jian Wen, Min Liang, Bo Su,Yanling Yin*. IGF-1 Alleviates NMDA-induced Excitotoxicity inCultured Hippocampal Neurons against Autophagy via the NR2B/PI3K-AKT-mTORPathway. Journal of Cellular Physiology.11(2014):1618-29. doi:10.1002/jcp.24607.
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